Pathologic human GR mutant incorporates a transdominant negative impact on the wild-form GR by inhibiting its translocation in to the nucleus: great importance with the ligand-binding domain for intracellular GR trafficking.[J Clin Endocrinol Metab. 2001]आदिराज प्रतिष्ठाण, मिरजगाव, ता. कर्ज�